Cell Signaling Technology

Product Pathways - Nuclear Receptor Signaling

Nuclear Receptor Antibody Sampler Kit #8595

kit   Nuclear   Nuclear receptors  

REACTIVITY

No. Size Price
8595T 1 Kit ( 8 x 20 µl ) ¥6,597.00 现货查询 购买询价
Kit Includes Quantity Applications Reactivity Homology† MW (kDa) Isotype
PPARγ (C26H12) Rabbit mAb #2435 20 µl W,IHC-P,IF-IC, H,M, R, 53, 57 Rabbit IgG
RARα Antibody #2554 20 µl W, M,R, 55 Rabbit
RXRα (D6H10) Rabbit mAb #3085 20 µl W,IP, H,M,R, 53 Rabbit IgG
Glucocorticoid Receptor (D8H2) XP® Rabbit mAb #3660 20 µl W,IP,IF-IC,F,ChIP, H,M,R,Mk, 80, 91, 94 Rabbit IgG
Androgen Receptor (D6F11) XP® Rabbit mAb #5153 20 µl W,IHC-P,IF-IC,F, H, 110 Rabbit IgG
Anti-rabbit IgG, HRP-linked Antibody #7074 100 µl W, Goat
Estrogen Receptor α (D8H8) Rabbit mAb #8644 20 µl W,IP,IF-IC,ChIP, H, 66 Rabbit IgG
Progesterone Receptor A/B (D8Q2J) XP® Rabbit mAb #8757 20 µl W,IP,IHC-P,IF-IC,F,ChIP, H, Mk, 90 (PR-A), 118 (PR-B) Rabbit IgG
RARγ1 (D3A4) XP® Rabbit mAb #8965 20 µl W,IP,IHC-P,IF-IC, H,M, R,Hm,B,Dg, 58 Rabbit IgG

Specificity / Sensitivity

Each antibody in the Nuclear Receptor Antibody Sampler Kit recognizes endogenous levels of total respective protein. Glucocorticoid Receptor (D8H2) XP® Rabbit mAb is predicted to cross-react with all known alternative translation start site generated isoforms of glucocorticoid receptor-α and glucocorticoid receptor-β, and does not cross-react with mineralocorticoid receptor. Progesterone Receptor A/B (D8Q2J) XP® Rabbit mAb does not cross-react with either the glucocorticoid receptor or the mineralocorticoid receptor. RARγ1 (D3A4) XP® Rabbit mAb is not predicted to cross-react with RARγ2, and does not cross-react with either RARα or RARβ. RXRα (D6H10) Rabbit mAb does not cross-react with either RXRβ or RXRγ. 
Nuclear Receptor Antibody Sampler Kit试剂盒中的每一个抗体都能识别各自内源性水平的总蛋白。预测Glucocorticoid Receptor (D8H2) XP® Rabbit mAb与所有已知的选择性翻译起始位点产生的糖皮质激素受体-α亚型和-β亚型可以发生交叉反应。该抗体不与盐皮质激素受体发生交叉反应。Progesterone Receptor A/B (D8Q2J) XP® Rabbit mAb不与糖皮质激素或盐皮质激素受体中的任何一个发生交叉反应。未预测到RARγ1 (D3A4) XP® Rabbit mAb与RARγ2发生交叉反应,但不与RARα或RARβ中的任何一个发生交叉反应。RXRα (D6H10) Rabbit mAb不与RXRβ或RXRγ中的任何一个发生交叉反应. 
 
 


Source / Purification

Polyclonal antibodies are produced by immunizing animals with a synthetic peptide corresponding to the sequence of human RARα protein. Polyclonal antibodies are purified by protein A and peptide affinity chromatography.Monoclonal antibodies are produced by immunizing animals with a synthetic peptide corresponding to residues near the amino terminal region of human androgen receptor protein, residues in the carboxy terminus of human ERα protein, residues surrounding Leu378 of human glucocorticoid receptor protein, residues surrounding Asp69 of human PPARγ protein, residues surrounding Tyr541 of human progesterone receptor protein, residues near the amino terminus of human RARγ1 protein, or residues near the amino terminus of human RXRα protein.该多克隆抗体通过用合成肽免疫动物制备,该合成肽是对应人RARα蛋白的序列。多克隆抗体由蛋白A和肽亲和层析纯化。该单克隆抗体通过用合成肽免疫动物制备,该合成肽是人雄激素受体蛋白靠近氨基末端区域的残基、人ERα蛋白靠近羧基末端的残基、人糖皮质激素受体蛋白亮氨酸(378位)附近的残基、人PPARγ蛋白天冬氨酸(69位)附近的残基、人孕酮受体蛋白酪氨酸(541位)附近的残基、人RARγ1蛋白靠近氨基末端的残基或人RXRα蛋白靠近氨基末端的残基。

Description

The Nuclear Receptor Antibody Sampler Kit provides an economical means to evaluate the presence and status of nuclear receptors. This kit contains enough primary antibody to perform four western blots per primary.Nuclear Receptor Antibody Sampler Kit提供了一种经济的评估核受体存在和状态的方法。该试剂盒包含足够的抗体,每种抗体可以完成四次western blots 实验。

Western Blotting

Western Blotting

After the primary antibody is bound to the target protein, a complex with HRP-linked secondary antibody is formed. The LumiGLO* is added and emits light during enzyme catalyzed decomposition.

Western Blotting

Western Blotting

Western blot analysis of extracts from NIH/3T3 and C6 cells, using RARα Antibody.

IHC-P (paraffin)

IHC-P (paraffin)

Immunohistochemical analysis of 3T3-L1 cells, undifferentiated (left) or differentiated (right) , using PPARγ (C26H12) Rabbit mAb.

IHC-P (paraffin)

IHC-P (paraffin)

Immunohistochemical analysis of paraffin-embedded mouse brown fat using PPARγ (C26H12) Rabbit mAb.

Western Blotting

Western Blotting

Western blot analysis of extracts from NIH/3T3 and 3T3-L1 cells (differentiated 6 days) using PPARγ (C26H12) Rabbit mAb.

IF-IC

IF-IC

Confocal immunofluorescent analysis of 3T3-L1 cells using PPARγ (C26H12A8) Rabbit mAb (red) showing nuclear localization in differentiated cells. Lipid droplets have been labeled with BODIPY 493/503 (green). Blue pseudocolor = DRAQ5™ (fluorescent DNA dye).

IHC-P (paraffin)

IHC-P (paraffin)

Immunohistochemical analysis of paraffin-embedded LNCaP (AR+, left) and DU145 (AR-, right) using Androgen Receptor (D6F11) XP® Rabbit mAb.

IHC-P (paraffin)

IHC-P (paraffin)

Immunohistochemical analysis of paraffin-embedded human prostate carcinoma using Androgen Receptor (D6F11) XP® Rabbit mAb.

IF-IC

IF-IC

Confocal immunofluorescent analysis of LNCaP (positive, left) and DU145 (negative, right) cells using Androgen Receptor (D6F11) XP® Rabbit mAb (green). Actin filaments have been labeled with DY-554 phalloidin (red).

Western Blotting

Western Blotting

Western blot analysis of extracts from LNCaP (AR+), MCF7 (AR+), PC-3 (AR-), and DU 145 (AR-) cells using Androgen Receptor (D6F11) XP® Rabbit mAb (upper) and β-Actin Antibody #4967 (lower).

Flow Cytometry

Flow Cytometry

Flow cytometric analysis of DU-145 cells (red) and LNCaP cells (blue) using Androgen Receptor (D6F11) XP® Rabbit mAb.

IF-IC

IF-IC

Confocal immunofluorescent analysis of HaCaT cells (positive, left) and Hep3B cells (negative, right) using RARγ1 (D3A4) XP® Rabbit mAb (green). Actin filaments were labeled with DY-554 phalloidin (red).

IHC-P (paraffin)

IHC-P (paraffin)

Immunohistochemical analysis of paraffin-embedded human skin using RARγ1 (D3A4) XP® Rabbit mAb.

IHC-P (paraffin)

IHC-P (paraffin)

Immunohistochemical analysis of paraffin-embedded human lung carcinoma using RARγ1 (D3A4) XP® Rabbit mAb.

IHC-P (paraffin)

IHC-P (paraffin)

Immunohistochemical analysis of paraffin-embedded cell pellets, HaCaT (positive, left) and Hep3B (negative, right), using RARγ1 (D3A4) XP® Rabbit mAb.

Western Blotting

Western Blotting

Western blot analysis of extracts from various cell lines using RARγ1 (D3A4) XP® Rabbit mAb.

Western Blotting

Western Blotting

Western blot analysis of extracts from 293T cells, either mock transfected (-) or transfected with a Myc/DDK-tagged cDNA expression construct encoding full-length human RARγ1 (hRARγ1-Myc/DDK, +), using RARγ1 (D3A4) XP® Rabbit mAb.

Western Blotting

Western Blotting

Western blot analysis of extracts from various cell lines using RXRα (D6H10) Rabbit mAb.

Isoform Specificity

Isoform Specificity

Western blot analysis of extracts from 293T cells, either mock transfected (-) or transfected with Myc/DDK-tagged cDNA expression constructs encoding full-length human RXRα (hRXRα; +), RXRβ (hRXRβ; +), or RXRγ (hRXRγ; +), using RXRα (D6H10) Rabbit mAb (upper) and DYKDDDDK Tag Antibody (Binds to same epitope as Sigma's Anti-FLAG® M2 Antibody) #2368 (lower).

IF-IC

IF-IC

Confocal immunofluorescent analysis of HeLa cells, grown in phenol red-free media containing 5% charcoal-stripped FBS for 2 days and either untreated (left) or dexamethasone-treated (100 nM, 2hr; right), using Glucocorticoid Receptor (D8H2) XP® Rabbit mAb (green). Actin filaments were labeled with DY-554 phalloidin (red).

Western Blotting

Western Blotting

Western blot analysis of extracts from various cell lines using Glucocorticoid Receptor (D8H2) XP® Rabbit mAb.

Western Blotting

Western Blotting

Western blot analysis of extracts from 293T cells, either mock transfected (-) or transfected with a construct expressing Myc/DDK-tagged full-length human glucocorticoid receptor-α (hGRα-Myc/DDK, +) or Myc/DDK-tagged full-length human mineralocorticoid receptor (hMR-Myc/DDK, +), using Glucocorticoid Receptor (D8H2) XP® Rabbit mAb (upper) and DYKDDDDK Tag Antibody (Binds to same epitope as Sigma's Anti-FLAG® M2 Antibody) #2368 (lower).

Chromatin IP

Chromatin IP

A549 cells were cultured in media with 5% charcoal-stripped FBS for 3 days and then either untreated (left panel) or dexamethasone-treated (100 nM, 1 hr; right panel). Chromatin immunoprecipitations were performed with cross-linked chromatin from 4 x 106 cells and 5 µl of Glucocorticoid Receptor (D8H2) XP® Rabbit mAb or 2 µl of Normal Rabbit IgG #2729 using SimpleChIP® Enzymatic Chromatin IP Kit (Magnetic Beads) #9003. The enriched DNA was quantified by real-time PCR using SimpleChIP® Human SLC19A2 Promoter Primers #7681, human MT2A promoter primers, and SimpleChIP® Human α Satellite Repeat Primers #4486. The amount of immunoprecipitated DNA in each sample is represented as signal relative to the total amount of input chromatin, which is equivalent to one.

Chromatin IP

Chromatin IP

Chromatin immunoprecipitations were performed with cross-linked chromatin from 4 x 106 MCF7 cells grown in phenol red free medium and 5% charcoal stripped FBS for 4 d then treated with β-estradiol (10 nM) for 45 minutes and either 5 μl of Estrogen Receptor α (D8H8) Rabbit mAb or 2 μl of Normal Rabbit IgG #2729 using SimpleChIP® Enzymatic Chromatin IP Kit (Magnetic Beads) #9003. The enriched DNA was quantified by real-time PCR using SimpleChIP® Human ESR1 Promoter Primers #9673, SimpleChIP® Human pS2 Promoter Primers #9702, and SimpleChIP® Human α Satellite Repeat Primers #4486. The amount of immunoprecipitated DNA in each sample is represented as signal relative to the total amount of input chromatin, which is equivalent to one.

IF-IC

IF-IC

Confocal immunofluorescent analysis of T-47D (PR positive, left) and MDA-MB-231 (PR negative, right) cells using Progesterone Receptor A/B (D8Q2J) XP® Rabbit mAb (green). Actin filaments were labeled with DY-554 phalloidin (red).

Western Blotting

Western Blotting

Western blot analysis of extracts from ER-positive cell lines (MCF7, T-47D, ZR-75-1) and ER-negative cell lines (SK-BR-3 and MCF 10A) using Estrogen Receptor α (D8H8) Rabbit mAb (upper) or β-Actin (D6A8) Rabbit mAb #8457 (lower).

IHC-P (paraffin)

IHC-P (paraffin)

Immunohistochemical analysis of paraffin-embedded cell pellets, T-47D (high PR, left), MCF-7 (low PR, middle) and MDA-MB-231 (PR negative, right), using Progesterone Receptor A/B (D8Q2J) XP® Rabbit mAb.

IHC-P (paraffin)

IHC-P (paraffin)

Immunohistochemical analysis of paraffin-embedded human infiltrating ductal breast carcinoma using Progesterone Receptor A/B (D8Q2J) XP® Rabbit mAb.

Western Blotting

Western Blotting

Western blot analysis of extracts from T-47D (PR positive) and MDA-MB-231 (PR negative) cells using Progesterone Receptor A/B (D8Q2J) XP® Rabbit mAb (upper) or GAPDH (D16H11) XP® Rabbit mAb #5174 (lower).

Western Blotting

Western Blotting

Western blot analysis of extracts from T-47D cells, grown for 48 hr in phenol red-free medium supplemented with 5% charcoal-stripped FBS and then treated with either a vehicle control (-) or promegestone (R5020, 100 nM, 16 hr; +), using Progesterone Receptor A/B (D8Q2J) XP® Rabbit mAb (upper) or GAPDH (D16H11) XP® Rabbit mAb #5174 (lower). Prolonged treatment of PR-expressing cells with R5020 is known to induce PR downregulation and hyperphosphorylation, which is reflected by slower migration on SDS-PAGE.

Chromatin IP

Chromatin IP

T-47D cells were cultured in phenol red-free media supplemented with 5% charcoal-stripped FBS for 48 hr and then either untreated (left panel) or promegestone-treated (R5020, 10 nM, 1 hr; right panel). Chromatin immunoprecipitations were performed with cross-linked chromatin from 4 x 106 cells and 5 µl of Progesterone Receptor A/B (D8Q2J) XP® Rabbit mAb or 2 µl of Normal Rabbit IgG #2729 using SimpleChIP® Enzymatic Chromatin IP Kit (Magnetic Beads) #9003. The enriched DNA was quantified by real-time PCR using SimpleChIP® Human FKBP51 Intron 5 Primers #7859, human E2F-1 proximal enhancer site #1 primers, and SimpleChIP® Human α Satellite Repeat Primers #4486. The amount of immunoprecipitated DNA in each sample is represented as signal relative to the total amount of input chromatin, which is equivalent to one.

IF-IC

IF-IC

Confocal immunofluorescent analysis of MCF7 (left) or SK-BR-3 (right) cells using Estrogen Receptor α (D8H8) Rabbit mAb (green). Actin filaments were labeled with DY-554 phalloidin (red).

Western Blotting

Western Blotting

Western blot analysis of extracts from various cell lines using Glucocorticoid Receptor (D8H2) XP® Rabbit mAb #3660.Western blot方法检测多个细胞系提取物,使用的抗体为Glucocorticoid Receptor (D8H2) XP® Rabbit mAb #3660.

Western blot analysis of extracts from various cell lines using RXRα (D6H10) Rabbit mAb #3085.Western blot方法检测多个细胞系提取物,使用的抗体为RXRα (D6H10) Rabbit mAb #3085.

Western blot analysis of extracts from AR-positive (LNCaP and MCF7) and AR-negative (PC-3 and DU 145) cell lines using Androgen Receptor (D6F11) XP® Rabbit mAb #5153 (upper) or β-Actin Antibody #4967 (lower).Western blot方法检测雄激素受体阳性细胞系(LNCaP 和MCF7)和雄激素受体阴性细胞系(PC-3和DU 145)提取物,使用的抗体为Androgen Receptor (D6F11) XP® Rabbit mAb #5153 (上图)或β-Actin Antibody #4967 (下图)。

Western blot analysis of extracts from NIH/3T3 and C6 cells using RARα Antibody #2554.Western blot方法检测 NIH/3T3细胞和C6细胞提取物,使用的抗体为RARα Antibody #2554.

Western blot analysis of extracts from NIH/3T3 and 3T3-L1 cells (differentiated 6 d) using PPARγ (C26H12) Rabbit mAb #2435.Western blot方法检测 NIH/3T3细胞和 3T3-L1细胞(分化6天)提取物,使用的抗体为PPARγ (C26H12) Rabbit mAb #2435.

Western blot analysis of extracts from PR-positive (T-47D) and PR-negative (MDA-MB-231) cell lines using Progesterone Receptor A/B (D8Q2J) XP® Rabbit mAb #8757 (upper) or GAPDH (D16H11) XP® Rabbit mAb #5174 (lower).Western blot方法检测 孕酮受体阳性细胞系(T-47D)和孕酮受体阴性细胞系(MDA-MB-231)提取物,使用的抗体为Progesterone Receptor A/B (D8Q2J) XP® Rabbit mAb #8757 (上图) 或GAPDH (D16H11) XP® Rabbit mAb #5174 (下图).

Western blot analysis of extracts from various cell lines using RARγ1 (D3A4) XP® Rabbit mAb #8965.Western blot方法检测多个细胞系提取物,使用的抗体为RARγ1 (D3A4) XP® Rabbit mAb #8965.

Western blot analysis of extracts from ER-positive (MCF7, T-47D, ZR-75-1) and ER-negative (SK-BR-3 and MCF 10A) cell lines using Estrogen Receptor α (D8H8) Rabbit mAb #8644 (upper) or β-Actin (D6A8) Rabbit mAb #8457 (lower).Western blot方法检测 雌激素受体阳性细胞系(MCF7, T-47D, ZR-75-1)和雌激素受体阴性细胞系(SK-BR-3和MCF 10A) 提取物,使用的抗体为Estrogen Receptor α (D8H8) Rabbit mAb #8644 (上图)或 β-Actin (D6A8) Rabbit mAb #8457 (下图).

Flow Cytometry

Flow Cytometry

Human whole blood was fixed, lysed, and permeabilized as per the Cell Signaling Technology Flow Alternate Protocol and stained using Glucocorticoid Receptor (D8H2) XP® Rabbit mAb (blue) compared to Rabbit (DA1E) mAb IgG XP® Isotype Control #3900 (red). Anti-rabbit IgG (H+L), F(ab')2 Fragment (Alexa Fluor® 488 Conjugate) #4412 was used as a secondary antibody. Samples were gated on CD3+ lymphocytes.

Flow Cytometry

Flow Cytometry

Flow cytometric analysis of MDA MB-231 cells (blue) and T47D cells using Progesterone REceptor A/B (D8Q2J) XP® Rabbit mAb. Anti-rabbit IgG (H+L), F(ab')2 Fragment (ALexa FLuor® 488 Conjugate) #4412 was used as a secondary antibody.

Background

Nuclear Receptors are transcription factors responsible for sensing bioactive molecules, including steroid and thyroid hormones. They are regulated by multiple posttranslational modifications, which in turn impacts their ability to regulate the expression of specific genes involved in the control of reproduction, development, and metabolism. 
 
 Androgen receptor (AR), a zinc finger transcription factor belonging to the nuclear receptor superfamily, is activated by phosphorylation and dimerization upon ligand binding (1). This promotes nuclear localization and binding of AR to androgen response elements in androgen target genes. AR plays a crucial role in several stages of male development and the progression of prostate cancer (2,3). 
 
 Estrogen receptor α (ERα), a member of the steroid receptor superfamily, contains highly conserved DNA binding and ligand binding domains (4). Through its estrogen-independent and estrogen-dependent activation domains (AF-1 and AF-2, respectively), ERα regulates transcription by recruiting coactivator proteins and interacting with general transcriptional machinery (5). 
 
 Glucocorticoid hormones control cellular proliferation, inflammation, and metabolism through their association with the glucocorticoid receptor (GR)/NR3C1, a member of the nuclear hormone receptor superfamily of transcription factors (6). 
 
 Peroxisome proliferator-activated receptor γ (PPARγ) is a member of the ligand-activated nuclear receptor superfamily and functions as a transcriptional activator (7). PPARγ is preferentially expressed in adipocytes, as well as in vascular smooth muscle cells and macrophages (8). Besides its role in mediating adipogenesis and lipid metabolism (8), PPARγ also modulates insulin sensitivity, cell proliferation, and inflammation (9). 
 
 Human progesterone receptor (PR) is expressed as two forms: the full length PR B and the short form PR A. PR A lacks the first 164 amino acid residues of PR B (10,11). Both PR A and PR B are ligand activated, but differ in their relative ability to activate target gene transcription (12,13). 
 
 Nuclear retinoic acid receptors (RARs) consist of three subtypes encoded by separate genes: α (NR1B1), β (NR1B2), and γ (NR1B3). For each subtype, there are at least two isoforms, which are generated by differential promoter usage and alternative splicing and differ only in their N-terminal regions. Retinoids, which are metabolites of vitamin A, serve as ligands for RARs (14). RARs function as ligand-dependent transcriptional regulators and are found to be heterodimerized with retinoid X receptors (RXRs). These transcriptionally active dimers regulate the expression of genes involved in cellular differentiation, proliferation, and apoptosis (15,16). 
 
 The human retinoid X receptors are encoded by three distinct genes (RXRα, RXRβ, and RXRγ) and bind selectively and with high affinity to the vitamin A derivative, 9-cis-retinoic acid. RXRs are type-II nuclear hormone receptors that are largely localized to the nuclear compartment independent of ligand binding. Nuclear RXRs form heterodimers with nuclear hormone receptor subfamily 1 proteins, including thyroid hormone receptor, retinoic acid receptors, vitamin D receptor, peroxisome proliferator-activated receptors, liver X receptors, and farnesoid X receptor (17).核受体是负责检测生物活性分子的转录因子,包括类固醇和甲状腺激素。它们受多种翻译后修饰的调控,反过来又影响参与生殖、发育和新陈代谢特定基因表达的自我调控能力。雄激素受体(AR),是锌指转录因子,属于核受体超家族,通过磷酸化和配体结合的二聚化被激活(1)。这促进了细胞核定位和雄激素靶基因中雄激素与应答元件的结合。 AR在男性发育和前列腺癌发展的几个阶段起着至关重要的作用(2,3)。雌激素受体α(ERα),是类固醇受体超家族的一员,含有高度保守的DNA结合域(DBD)和配体结合域(LBD)(4)。ERα通过其雌激素非依赖性和雌激素依赖性激活域(分别为AF-1和AF-2),调节转录招募共激活因子蛋白和与一般转录机制的相互作用(5)。糖皮质激素通过与转录因子的细胞核激素受体超家族成员糖皮质激素受体(GR)/ NR3C1联合,控制细胞增殖、炎症和代谢(6)。 过氧化物酶体增殖物激活受体γ(PPARγ)是配体激活的核受体超家族成员,作为转录激活剂起作用(7)。 PPARγ优先在脂肪细胞、血管平滑肌细胞和巨噬细胞表达(8)。除此之外,它在介导的脂肪细胞分化和脂质代谢中起作用(8),PPARγ调节胰岛素敏感性、细胞增殖和炎症(9)。 人孕酮受体(PR)表现为两种形式:全长的孕酮受体B和短的孕酮受体A。PR A缺少PR B的第164个氨基酸残基(1,2)。PR A和PR B都是配体激活,但其激活靶基因转录的相对能力是不同的(12,13)。
维甲酸核受体(RARs)包括三个亚型,这三个亚型由分别由α (NR1B1), β (NR1B2), 和γ (NR1B3)编码.每种亚型至少有两种形式,通过差动启动子使用和选择性剪接产生,不同之处仅在于它们的N-端区域。维甲酸是维生素A的代谢产物,作为维甲酸受体的配体(14)。维甲酸受体以配体依赖的转录调节起作用,发现其与维甲酸X受体(RXRs)形成异源二聚体。这些转录活性二聚体调节参与细胞分化、增殖、凋亡的基因表达(15,16)。 
人视黄醇X受体(RXRs)由三种不同的基因(RXRα,RXRβ,RXRγ)编码,并与具有高亲和力的维生素A衍生物,9-顺-视黄酸选择性结合。 RXRs是II型核激素受体,主要位于核室,非依赖性配体结合。核RXR与核激素受体亚家族蛋白形成异二聚体,包括甲状腺激素受体、维甲酸受体、维生素D受体、过氧化物酶体增殖物活化受体、肝脏X受体、法尼酯X受体(17)。

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U.S. Patent No. 5,675,063.

Cell Signaling Technology is a trademark of Cell Signaling Technology, Inc.

Cell Signaling Technology® is a trademark of Cell Signaling Technology, Inc.

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